Cell Line

hPSCreg name WAe009-A-24
Cite as:
WAe009-A-24 (RRID:CVCL_YN82)
Cell line type Human embryonic stem cell (hESC)
Similar lines No similar lines found.
Last update 4th November 2019
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Generator Monash Institute of Pharmaceutical Sciences (MIPS)
Owner Monash Institute of Pharmaceutical Sciences (MIPS)
Derivation country Australia

External Databases

BioSamples SAMEA6324522
Cellosaurus CVCL_YN82
Wikidata Q98134748

General Information

* Is the cell line readily obtainable for third parties?
Subclone of

Donor Information

General Donor Information

Sex female
Ethnicity N/A

Phenotype and Disease related information (Donor)

Diseases A disease was diagnosed.
  • HD
  • Huntington disease
  • Huntington's chorea
Family history NO
Is the medical history available upon request? NO
Is clinical information available? NO

Karyotyping (Donor)

Has the donor karyotype been analysed?
Karyotyping method: Molecular karyotyping by SNP array

Other Genotyping (Donor)

Is there genome-wide genotyping or functional data available?

Donor Relations

Other cell lines of this donor

External Databases (Donor)

BioSamples SAMEA7768918


Also have a look at the ethics information for the parental line WAe009-A .
Is there an MTA available for the cell line? No

hESC Derivation

The source cell information can be found in the parental cell line WAe009-A.

Culture Conditions

Surface coating Matrigel/Geltrex


No characterisation data could be found for this subclone. Please open parental cell line WAe009-A .


Karyotyping (Cell Line)

Other Genotyping (Cell Line)

Genetic Modification

Genetic modifications not related to a disease
CX3CR1 (target)
Gene knock-in
This line is engineered to include a genetic reporter of CX3CR1 expression which results in expression of tdTomato and nanoluciferase in series with CX3CR1 expression. The construct also includes a neomycin resistance cassette flanked by FRT sites. One allele of the CX3CR1 gene contains the following construct targeted to the stop codon: IRES-tdTomato-T2A-Nanoluc-pA-FRT-neo-FRT.